![]() ![]() Often if P waves are visible, they appear in retrograde fashion as inverted P waves following QRS waves in II, III, and aVF. P waves can be difficult to see and often are buried in the QRS complex. AVnRT can be distinguished from other tachyarrhythmias as often appears as regular tachycardia with HR ~160 bpm (though can range ~140-280 bpm). Although we commonly say SVT (and are unlikely to change), SVT can refer to any tachyarrhythmia arising above the AV Node. ![]() This is what we think of when we say SVT. Therefore, the treatment revolves around the treatment of the underlying cause. fever, hypovolemia, PE, stress/anxiety, exercise), and not due to a cardiac issue. Sinus tachycardia is often caused by a systemic issue (e.g. Sinus is the default rhythm of the heart and originates from the SA node. ![]() Just a quick review that sinus is defined as a QRS complex preceded by a P wave. Furthermore, the regular rhythms can be broken down into “AV Nodal dependent” and “AV nodal independent” rhythms, which can help us remember the treatments as well. The one that I find the most useful is breaking them into regular and irregular rhythms. There are several different ways to break down the differential for narrow complex tachycardia. Although we will not delve too deep into antiarrhythmics, it is important to note that electricity is safe in all unstable rhythms no matter the etiology. The goal of this blog is to run through this differential and give some methods to differentiate the rhythms. It includes rhythms such as sinus tachycardia, AVnRT, AVRT, atrial flutter, ectopic atrial tachycardia (EAT), atrial fibrillation, atrial flutter, and multifocal atrial tachycardia (MAT). The differential for narrow complex tachycardia is extremely important as it is the most commonly seen abnormal EKG in the emergency department. ![]()
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